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Deterioration of Articular Cartilage Caused by Continuous Compression in a Moving Rabbit Joint A LIGHT AND ELECTRON MICROSCOPIC STUDY
THOMAS L. GRITZKA; LOUIS R. FRY; ROY L. CHEESMAN; ANGELA LAVIGNE
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3025 North Vancouver Avenue, Portland, Oregon 97227 Department of Orthopaedics, University of Washington, Seattle, Washington Lloyd Medical Engineering Laboratory, 5463 South Othello Street, Seattle, Washington 98118
1973 by The Journal of Bone and Joint Surgery, Incorporated
The Journal of Bone & Joint Surgery.  1973; 55:1698-1720 
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Abstract

Extension springs were placed across the elbow joints of mature but not senescent rabbits in a manner which produced compression but allowed the joint to move. Compression was maintained for from one to thirty-two days. Within the range of compression used, the severity of cartilage damage correlated with the duration rather than the magnitude of the compression. As the arthritic lesion developed, the cartilage matrix underwent fibrillation and eventually was eroded away entirely, while a proliferative response was evoked at the joint margin. The chrondrocytes became pyknotic and then, in the end stages, were compressed into linear masses devoid of internal structure.

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    Accreditation Statement
    These activities have been planned and implemented in accordance with the Essential Areas and policies of the Accreditation Council for Continuing Medical Education (ACCME) through the joint sponsorship of the American Academy of Orthopaedic Surgeons and The Journal of Bone and Joint Surgery, Inc. The American Academy of Orthopaedic Surgeons is accredited by the ACCME to provide continuing medical education for physicians.
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