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Transverse Deficiency   |    
Molecular Mechanisms to Improve Nerve Regeneration Following Damage to the Immature Peripheral Nervous System
Peter Bannerman, PhD1; Michelle A. James, MD1
1 Institute for Pediatric Regenerative Medicine (P.B.) and Department of Orthopaedic Surgery (M.A.J.), Shriners Hospital for Children Northern California, 2425 Stockton Boulevard, Sacramento, CA 95817. E-mail address for P. Bannerman: pgbannerman@ucdavis.edu
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Disclosure: In support of their research for or preparation of this work, one or more of the authors received, in any one year, outside funding or grants in excess of $10,000 from the National Institutes of Health (Grant #RO1 NS025044). Neither they nor a member of their immediate families received payments or other benefits or a commitment or agreement to provide such benefits from a commercial entity.

The Journal of Bone and Joint Surgery, Inc.
J Bone Joint Surg Am, 2009 Jul 01;91(Supplement 4):87-89. doi: 10.2106/JBJS.I.00279
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Extract

Defects in human limb growth and development are typically attributed to genetic and environmental factors and less commonly to the role of the nervous system. Chronic lack of neural activity, whether due to disuse, environmental factors, or, most commonly, nerve trauma, adversely affects muscle, bone, and joint development. The most prominent example of this problem is disruption of the brachial plexus during birth, a condition that can affect the entire upper limb.
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    Accreditation Statement
    These activities have been planned and implemented in accordance with the Essential Areas and policies of the Accreditation Council for Continuing Medical Education (ACCME) through the joint sponsorship of the American Academy of Orthopaedic Surgeons and The Journal of Bone and Joint Surgery, Inc. The American Academy of Orthopaedic Surgeons is accredited by the ACCME to provide continuing medical education for physicians.
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